How is ulerythema ophryogenes treated?
Treatment for ulerythema ophryogenes has been unsatisfactory. Although local applications of salicylic acid, resorcinol, and the mercurials have been advocated, few, if any, cases respond. Recently, a patient was treated with hydrocortisone ointment with spectacular results.
What is eyebrow keratosis?
Keratosis pilaris atrophicans faciei is an uncommon form of keratosis pilaris with scar-like follicular depressions and loss of hair particularly in the eyebrows [1]. This results in atrophy and permanent loss of hair in the affected areas. Keratosis pilaris is also called ulerythema ophryogenes.
What causes keratosis pilaris rubra Faceii?
What causes it? Keratosis pilaris is, in most cases, a genetic condition that runs in families. Hair follicle openings become plugged with a build-up of keratin, a protein that lines the hair and skin. Some of the bumps may contain an ingrown hair.
How do you treat eyebrows for keratosis pilaris?
Treatment with topical keratolytic agents, such as lactic acid, glycolic acid, salicylic acid, tretinoin, vitamin D3 analogue, or urea in combination with a topical corticosteroid or oral retinoids, has been used with some success.
What is similar to keratosis pilaris?
Keratosis pilaris may resemble the following uncommon skin conditions: Lichen spinulosus. Pityriasis rubra pilaris. Ulerythema ophryogenes (ulerythema)
What causes lichen Spinulosus?
The cause of lichen spinulosus is unknown. It appears to be an abnormal reaction pattern of follicles to various triggers. Some people relate LS to malnutrition, specifically vitamin A deficiency. Others think it arises after exposure to toxins.
How do you treat keratosis pilaris rubra?
Treatments for KP commonly include emollients, keratolytic agents, topical corticosteroids, and topical retinoids, but these are often ineffective in diminishing the appearance of KP. Erythema is sometimes present in KP, but is usually mild and limited to the perifollicular skin.
What are follicular plugs?
Follicular keratotic plugs are a marker of DLE and were originally described as a sign of early and active lesions and not in areas of scarring or healed skin. [3] They correlate with the hyperkeratosis and the plugging of follicular ostia with keratotic material.